Even if the 2-D model is incomplete, ask yourself: according to the criteria we use for the lifts, is the sumo deadlift as effective as the conventional for general strength training?
Without a doubt, it uses less muscle mass by removing a significant moment arm from the spinal erectors (which effectively operate entirely in the sagittal plane and can be decently modeled in 2D).
It shortens the ROM (potentially dramatically, depending on the lifter)
The wider the stance, the more force is directed in the frontal plane, not lifting the barbell, limiting total weight on the bar and therefore total systemic stress.
Basically, sumo's sole value is in how it shifts the limiting factor in the lift from the back to the legs or hips... but since we're trying to train the back in the deadlift, that defeats our purpose. Sumo is still useful for some competitive lifters because it spares a back that's been cooked by 2 limit squats, but it's inferior for general strength development based on our criteria.
I'm not actually sure what you're saying here. Even within the limitations of a 2-D framework looked at from the side, it's perfectly easy to see why touching the bar too low limits optimal strength development because it dramatically increases the moment arm on the shoulders while (pretty much) cutting the pecs out of the equation. So... we agree???
EMG studies aren't necessarily useless, but what do they tell you? If the back is cut out of the equation and the knee/hip has to bear a greater share of the load (and they weren't maximally taxed in the first place), I wouldn't be surprised if the EMG for the knee or hip increased. That doesn't mean it's a great strength exercise for our purposes, though... because this ' already presupposed that we're cutting out the back, something we don't want.
The article you cited has been addressed in the subforums on this site several times.
The erectors are not an agonist at all. They are a stabilizer. They act isometrically to hold the spine in place so that the agonists can complete the act of hip and knee extension that define the lift. Your article author has invented his own arbitrary definitions of 'agonist' and 'synergist' and then claimed, using these definitions, that the erectors are 'agonists' (seriously: compare his definitions with a physiology text).
His argument that a shortening of the height between the sacrum and c-7 proves the erectors are acting concentrically ignores the fact that there are compressible structures (the discs of the spine) that shrink under load and could explain this change. He also slips into the common trap that a muscle 'acts' isometrically. It contracts. Period. If its force of contraction is exactly enough to counter the force of another muscle or an external weight so that the muscle does not change length (and the joint the muscle is supposed to rotate does not change angle), it is isometric, regardless of how difficult or painful it is (see his ab-wheel analogy). That's not to say it's a pointless article: it's great to find an article that appreciates the importance of the back and doesn't simply tell the lifter to be "as vertical as possible," but it's not technically correct.
As for the cause of hypertrophy, there's a lot more than lengthening/shortening under load. The mechanical stress itself, for instance, initiates a paracrine hormonal response. You could also be right, that form faults give the thoracic erectors extra concentric work, but it's not necessary to explain hypertrophy.
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Callador... I'm pretty sure you have a kitten meme for literally every conceivable situation. Bravo.