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Thread: The relationship between cortisol, carbs, testosterone and resistance training ?

  1. #11
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    Quote Originally Posted by Robert Santana View Post
    Wrong. You must have a high ratio of NADH:NAD+ not NADH:FADH2 ratio to have extensive H2O2 efflux (I.e. this occurs when you consume excessive amounts of alcohol). The other condition would be a high proton conductance in conjunction with reduced CoQ and no ATP synthesis. Otherwise under normal conditions H2O2 efflux is very low.
    Yes I agree with NADH:NAD+ ratio which is the redox couple at Complex I, but I was referring to MORE NADH and LESS FADH2 input from carbs versus fats. Higher NADH from carbs WOULD lower NAD+ which in turn lowers redox potential leading to more ROS generation. That's what I meant.

    Ketosis raises NAD+ levels which raises redox and lowers ROS. That's what I'm saying my whole point is that fat burning generates less ROS than glycolysis.

  2. #12
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    Again, under normal conditions superoxide production is marginal and so is oxidative stress. I discussed the abnormal conditions where this would be an issue.

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