Data?
Member
Why do power lifters and body builders on steroids occasionally…
Why do power lifters and body builders on steroids occasionally rip their tendons and are people on TRT at increased risk of tendon damage? Also what is mechanism by which the increase rate of damage occurs amongst these groups who are on steroids?
TMPHBITEU
Data?
Member
Do not feed.
TMPHBITEU
I want to see the boy's data.
Member
You can't ask a question about an unknown topic, then immediately ask another question that's based on that topic being known. That implies that you already know the answer to the previous question.
Member
Never heard about all that, but I ruptured my achilles tendon playing on a "warped wall" on Memorial day weekend and wanted to thank Rip for the info on his recovery process that he shared here. I'm like 13 weeks out and almost back to squatting what I was pre-injury (still can't fully "calf raise" on my injured leg, but I'm getting close).
My tear was predisposed by the antibiotic Clindamycin, which is low on the list of antibiotics that are linked. My podiatrist was well aware of that connection (Cipro is super bad about this). I would conjecture that steroids would do the opposite of what the OP asked about. More people should probably be informed about the antibiotic thing though. Thanks again for your sharing your experience with your achilles rupture, Rip. This board was incredibly helpful from the psychological perspective in my recovery.
Member
TMPHBITEU
From the paper:
A very real possibility is that men who use AAS (note that this is not the same thing as TRT) tend to do things that get their tendons ruptured. To their credit, the authors discuss the limitations of the study. You should too.The causes of AAS-associated tendon rupture are still incompletely understood. Two alternative (and not mutually exclusive) hypotheses should be considered. One possibility is that AAS use has little or no deleterious effect on tendons themselves, but merely causes massive hypertrophy of muscles without causing any corresponding strengthening of the associated tendons. Thus, the muscle may simply become too strong for its tendon, increasing the possibility of rupture in response to a sudden stress. Alternatively, it is possible that high doses of AAS, perhaps in conjunction with intense muscular exercise, may damage the structure of the tendons themselves, making them more vulnerable to rupture even in the absence of excessive stress. Evidence favoring the latter hypothesis comes from various animal studies, which have typically found that AAS exposure, usually in conjunction with exercise, led to collagen dysplasia, causing tendons to become stiffer and less flexible, with an increased crimp angle and earlier liability to failure.14, 21, 30–32, 34–39, 52 However, one human study using electron microscopy found no evidence of collagen fibril ultrastructural abnormalities in the ruptured tendons of two AAS users as compared to two non-AAS-using controls.9 Another recent study found no significant difference in maximal strain and toe limit strain in the patellar tendons of 8 long-term AAS users as compared to 8 experienced weightlifters reporting no AAS use, also arguing against the hypothesis of changes in collagen crimp pattern associated with AAS use.47 Thus the evidence for a direct toxic effect of AAS on human tendons remains somewhat inconclusive.
✓ Member
You are aware that a cross-sectional study is merely correlational and can't imply causality? And that data from a single study can not be extrapolated to the entire world? That you would need to show the effect in repeated and separate high quality research studies to demonstrate an effect, and even then it is only suggestive? That there are probably hundreds or thousands of factors that we can't control when looking through such studies? And that most are of poor quality or biased by the researchers? And research would only make sense when considered alongside many years of practical experience by experts in the field that work with actual people (coaches, trainers, etc.)?Originally Posted by 5867-Rich
This study is a cohort study - meaning its susceptible to factors that affect the entire cohort. It's correlational data, not experimental, so you can't extrapolate further. It's just a paper to draw attention to an area where other researchers can further research, or people with actual experience with athletes can weigh in.
Honestly, kids these days find a random study online and take it as the Bible of the way of the universe. You need to learn to read these things with a little more skepticism.
Member
In ref. to their first theory, tendons are avascular, which would make it rather difficult for them to keep up with steroid aided muscular advancement.
As they say nothing conclusive so far but can I ask you go out on a limb?
RUSSIAN powerlifter Alexander Sedykh fractured both knees while attempting to squat 400kg - YouTube
Is the above knee snap relevant to their theory of tendon development being slower than muscular development?
I realise there could be other causes but what is most likely in your mind?
What about pec tendon detachment same question?
How many anecdotes are required to draw an association between steroid use and ligaments letting go or do you think they’re not related?
Posting Permissions